OK:
I forget now who originally posted these on this forum, but I keep it in my archives because it offers a nice 'linear' progression of testing a methodology and then applying it - I have posted this more than a dozen times for creationists who claim that there is no evidence for evolution:
The tested methodology:
Science 25 October 1991:
Vol. 254. no. 5031, pp. 554 - 558
Gene trees and the origins of inbred strains of mice
WR Atchley and WM Fitch
Extensive data on genetic divergence among 24 inbred strains of mice provide an opportunity to examine the concordance of gene trees and species trees, especially whether structured subsamples of loci give congruent estimates of phylogenetic relationships. Phylogenetic analyses of 144 separate loci reproduce almost exactly the known genealogical relationships among these 24 strains. Partitioning these loci into structured subsets representing loci coding for proteins, the immune system and endogenous viruses give incongruent phylogenetic results. The gene tree based on protein loci provides an accurate picture of the genealogical relationships among strains; however, gene trees based upon immune and viral data show significant deviations from known genealogical affinities.
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Science, Vol 255, Issue 5044, 589-592
Experimental phylogenetics: generation of a known phylogeny
DM Hillis, JJ Bull, ME White, MR Badgett, and IJ Molineux
Department of Zoology, University of Texas, Austin 78712.
Although methods of phylogenetic estimation are used routinely in comparative biology, direct tests of these methods are hampered by the lack of known phylogenies. Here a system based on serial propagation of bacteriophage T7 in the presence of a mutagen was used to create the first completely known phylogeny. Restriction-site maps of the terminal lineages were used to infer the evolutionary history of the experimental lines for comparison to the known history and actual ancestors. The five methods used to reconstruct branching pattern all predicted the correct topology but varied in their predictions of branch lengths; one method also predicts ancestral restriction maps and was found to be greater than 98 percent accurate.
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Science, Vol 264, Issue 5159, 671-677
Application and accuracy of molecular phylogenies
DM Hillis, JP Huelsenbeck, and CW Cunningham
Department of Zoology, University of Texas, Austin 78712.
Molecular investigations of evolutionary history are being used to study subjects as diverse as the epidemiology of acquired immune deficiency syndrome and the origin of life. These studies depend on accurate estimates of phylogeny. The performance of methods of phylogenetic analysis can be assessed by numerical simulation studies and by the experimental evolution of organisms in controlled laboratory situations. Both kinds of assessment indicate that existing methods are effective at estimating phylogenies over a wide range of evolutionary conditions, especially if information about substitution bias is used to provide differential weightings for character transformations.
We can hereby CONCLUDE that the results of an application of those methods have merit.
Application of the tested methodology:
Implications of natural selection in shaping 99.4% nonsynonymous DNA identity between humans and chimpanzees: Enlarging genus Homo
"Here we compare ≈90 kb of coding DNA nucleotide sequence from 97 human genes to their sequenced chimpanzee counterparts and to available sequenced gorilla, orangutan, and Old World monkey counterparts, and, on a more limited basis, to mouse. The nonsynonymous changes (functionally important), like synonymous changes (functionally much less important), show chimpanzees and humans to be most closely related, sharing 99.4% identity at nonsynonymous sites and 98.4% at synonymous sites. "
Mitochondrial Insertions into Primate Nuclear Genomes Suggest the Use of numts as a Tool for Phylogeny
"Moreover, numts identified in gorilla Supercontigs were used to test the human–chimp–gorilla trichotomy, yielding a high level of support for the sister relationship of human and chimpanzee."
A Molecular Phylogeny of Living Primates
"Once contentiously debated, the closest human relative of chimpanzee (Pan) within subfamily Homininae (Gorilla, Pan, Homo) is now generally undisputed. The branch forming the Homo andPanlineage apart from Gorilla is relatively short (node 73, 27 steps MP, 0 indels) compared with that of thePan genus (node 72, 91 steps MP, 2 indels) and suggests rapid speciation into the 3 genera occurred early in Homininae evolution. Based on 54 gene regions, Homo-Pan genetic distance range from 6.92 to 7.90×10−3 substitutions/site (P. paniscus and P. troglodytes, respectively), which is less than previous estimates based on large scale sequencing of specific regions such as chromosome 7[50]. "
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CONCLUSION:
This evidence lays out the results of employing a tested methodology on the question of Primate evolution. No molecular phylogenetic analyses have produced results that counter the results of these.
Feel free to provide evidence for magic creation of a man from dust 6-10,000 years ago.
Imagine a creationist that can make a single argument without PLAGIARIZING creationist hacks.
It has to be imagined, because I have yet to encounter a creationist that can actually make their own arguments without at least paraphrasing other creationists.
Tell us all - did you plagiarize that crap from THIS CLOWN, or THIS ONE, who quoted the other? Reported.
I am pretty sure that you do not understand any of that, so I will debunk it for the sake of those that can in a subsequent post.
Sure. Until there is a good reason to think otherwise - and no, ReMine and Batten's dopey slogans do not count.
Unless you are Donny Batten, YEC, those are NOT "your" numbers.
I guess you folks like to ignore that many held that Haldane's dilemma was never really a dilemma:
Warren Ewens interview:
AP: The mention of Crow brings up a further question I had. Were many biologists concerned about the problems of genetic load and mutation load at the time you began your graduate career?
WE: [...]
A second form of the load concept was introduced by the British biologist-mathematician Haldane who claimed, in 1957, that substitutions in a Darwinian evolutionary process could not proceed at more than a certain comparatively slow rate, because if they were to proceed at a faster rate, there would be an excessive “substitutional load.” Since Haldane was so famous, that concept attracted a lot of attention. In particular, Crow and Kimura made various substitutional load calculations around 1960, that is at about that time that I was becoming interested in genetics.
Perhaps the only disagreement I ever had with Crow concerned the substitutional load, because I never thought that the calculations concerning this load, which he and others carried out, were appropriate. From the very start, my own calculations suggested to me that Haldane’s arguments were misguided and indeed erroneous, and that there is no practical upper limit to the rate at which substitutions can occur under Darwinian natural selection.
I already explained the problem with that dopey argument you plagiarized - you are just ignoring, like most dishonest creationists do.
First, Donny gets the argument wrong.
Second, he sets up a strawman scenario.
Third, he doesn't seem to understand population genetics.
Fourth, he did not address the questions I asked you, such that his strawman even had hypothetical merit:[/COLOR]
1. What traits the human-chimp ancestor had in the first place
2. how many mutations would have been needed in order to get a distinctly human trait - say, upper limb proportion - from the LCA of humans and chimps
2a. How you discovered what the ancestral state was, seeing as we do not know what the exact ancestral taxon was
2b. how you determined the number of beneficial mutations needed to produce that change
etc.
If you do not know what traits the ancestor had, and you have no idea how many mutations would have been "required" top alter those traits into the traits modern humans have, how on earth can you or Donny Batten or electrician ReMine possibly declare that number - or ANY number of mutations - to be 'too few' and be taken seriously?