Those are duplicated genes Jerry - all the 'new' genes are genes that the parents already have. Duplication alone is not going to get evolution from the first simple organism to all the other organisms. Simply buying the same newspaper twice means you can have twice as much content but exactly the same information
No, duplication alone is not enough. There must then be cxhanges in the base-pair order on either of the two genes. This is called "mutation" and you have stipulated that it does indeed happen.
Buying tomorrows paper is indeed new information, even if the format was copied from yesterdays.
The parent produces exactly the same proteins as the DS - the only difference is quantity of the proteins and perhaps regulation (switching on and off of the genes).
Chromosome 34 interferes with X-inactivation, creating a new morphology wich does not occur in any circmustance without it. Welcome to evolution.
The sickle cell gene forms a defective product - one that does not interact properly with the biochemical environment in which it is in. This is not a mechanism by which the first simple organism can evolve to all others - we are simply observing mutations damaging genes. Damage alone doesn't equal progress.
Defective is subjective and entirely irrellevent. The gene is more useful than harmful in areas with high malaria.
I could equally argue that the non-sickle-cell gene creates a defect by where the body is vunerable to destruction via malaria. The point is that the sickle cell gene is "new information" on an existing gene that does a "new thing". It's "novel".
Your only real counter is that it occurs in a spot that already exists (IOW, you would be arguing that new spots cannot be made); downs syndrome proves that new spots can be made.
I think you will find that the mutations are occurring in what you would call 'junk DNA' - sections of the DNA which don't code for the proteins - ie. sections of DNA which are not genes.
Many are... and that fact would change the frequency of gene changes. It would have no impact at all on the fact that genes change.
Do you agree that functioning genes and mutate in to other functioning genes (if not, how do you explain sickle-cell anemia?).
Do you agree that functioning genes can be copied and still function while their originals are also functioning? (If not, how do you explain downs syndrome).
If both of those are "yes", under what pretext do you assert that a compied, functioning gene is unable to mutate jus like an "uncopied" functioning gene to produce a changed copied functioning gene ("novel new")? It's the only scenerio I've not orced an actual example down our throat on, but it's one you've offered ZERO support against.
No it is not - but the theory of evolution doesn't merely rely on the genetic side of things. But i am approaching this from that side to show that there is a difference between microevolution and macroevolution.
Of course not, it goes into biology and reproduction... 99% of them are evolutionsts to. Explain that.
It is you rather than me that does not have the mechanism showing that macroevolution can occur - because there are no examples of it, the mechanism has never been observed to happen.
"macroevolution" is a fictitious and (you admitted yourself) undefined standard that means whatever the creationist needs it to mean in order for there to not be an example.
Once upon a time, "kind" ment "species"... but then we started showing examples of speciation and it moved, and moved, and moved again.
I told you early in this thread you would do exactly that with "new gene", having moved from "information not contained in either parent that functions" to "base pairs , that don't match other base pairs, appearing sudddenly in a new location, creating a protien which is no created anywhere else, which effects morphology, in a "positive" way, and does not cause sterility"
I show you one of those, and you'll add another criteria and another untli there isn't an example. It's the exact same thing creatinsts do with "missing links", it's the exact same thing they do with "kind", and you are doing it now with "novel new".
The analogy I made early on is very accurate here. You are asserting that it's impossible to write Harry Potter with a pencil because no one has seen it done.
The mechanism that would have to be observed would be mutation causing a new gene, which had a new function that was biochemically advantageous to the organism and fitted in with all the existing biochemical pathways in the organism.
And at this point you are demanding it to occur all simultaniously, which isn't what happenes.
How do you explain that there isn't a single number of functioning genes in the human species? Different people have different numbers.
You also never told me if a chimp could become a human with microevoltuion. We don't appear to have any more genes than they do.
Take the feathers as an example - genes had to be formed that previously didn't exist to control and bring that whole process of forming feathers into effect. We do not observe a mechanism occuring that could have lead to this.
Feathers is a mutation of hair (or vice-versa). It's a change in the genes that are responsable for scales, hair, and feathers. It's not "novel new" by the standard you've put up.
Of course, the fossil record shows animals like the dromaeosaur with pre-feathers in non-birds.