No, you provided a guess as to why cholesterol is found at the site.
Provide a link to any conclusive scientific evidence of causality, and then you can say that you " explained precisely how cholesterol contributes to artherosclerosis".
Scientic-sounding guesses are not science.
This part was not a guess. This is the mechanism by which cholesterol causes atherosclerosis, as taught by the ASCP (American Society for Clinical Pathology).
LDL particles and cholesterol play a critical role in atherogenesis. LDL particles are small enough to enter the intima, the innermost lining of the arterial wall. High levels of LDL particles translate into a dose-related increase in transport of the LDL-C component into the intima. The cells in the arterial wall have no ability to metabolize cholesterol. In addition, LDL-C may become trapped by binding with proteoglycans, making LDL-C more susceptible to the processes involved in the formation of plaque.
Oxidation, a primary process involved in plaque formation, stimulates inflammation, which attracts monocytes into the arterial wall. The monocytes differentiate into macrophages and scavenge the modifi ed LDL. As time passes, the LDL-macrophagecomplex transforms into foam cells, leading to plaque formation.1,3,4
See the 1, 3, and 4, at the end of the quote? That denotes the following sources:
1. Brunzell JD, Davidson M, Furberg CD,
et al. Lipoprotein management in patients
with cardiometabolic risk: consensus
conference report for the American Diabetes
Association and the American College of
Cardiology Foundation. J Am Coll Cardiol.
2008;51(15):1512-1524.
3. National Cholesterol Education Program
(NCEP) Expert Panel on Detection,
Evaluation, and Treatment of High Blood
Cholesterol in Adults (Adult Treatment
Panel III). Third Report of the National
Cholesterol Education Program (NCEP)
Expert Panel on Detection, Evaluation, and
Treatment of High Blood Cholesterol in
Adults (Adult Treatment Panel III) final
report. Circulation. 2002;106:3143-3421.
4. Nordestgaard BG, Benn M, Schnohr P,
et al. Nonfasting triglycerides and risk
of myocardial infarction, ischemic heart
disease, and death in men and women.
JAMA. 2007;298:299-308.